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4 Therapeutic interventions and platforms for modulating neuroinflammation Both local and systemic biomaterial-based strategies have emerged to modulate neuroinflammation in CNS disorders, offering complementary advantages in delivery precision, immune cell targeting, and sustained release

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For the last three years, this active targeting strategy for nano-drug targeted delivery systems for colorectal cancer has implemented the receptor-ligand binding strategy, which included many increasingly expressed CRC receptors, such as a folate receptor, CD44, epithelial cell adhesion molecule (EpCAM), CD133, nucleolin, EGFR, v3 integrin receptor, MUC1, P-selectin, SSTRs, glucocorticoid receptor, sigma-2 receptors, CXCR4+, checkpoint kinase 2, lipoprotein receptor-related protein-1, mannose receptor, carcinoembryonic antigen, hyaluronic acid receptor, and N-acetyl-D-glucosamine