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AIFM2 blocks ferroptosis independent of ubiquinol metabolism

1A), alone and in combination with Telaglenastat (CB-839), a selective glutaminase inhibitor of both splice variants of glutaminase (KGA and GAC) with good oral bioavailability [63, 64], currently in phase II clinical trials (NCT04265534)

In the cardiovascular context, DAMPs release from ferroptotic cardiomyocytes or endothelial cells activates resident macrophages, recruits circulating monocytes and neutrophils, and stimulates the production of inflammatory cytokines such as tumor necrosis factor- (TNF-), interleukin-1 (IL-1), and interleukin-6 (IL-6)

In some clinical protocols, these peptides are combined or alternated