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Central to the molecular machinery that links synaptic activity with such plastic changes is the Neuronal Per Arnt Sim domain protein 4 (NPAS4), a brain-specific basic helix-loop-helix transcription factor, whose expression is tightly regulated by synaptic activity and postsynaptic calcium influx (Flavell and Greenberg, 2008)

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However, we can infer that both the gradient and homogenous alloyed QDs display high degrees of monodispersity