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Modulation of oxidative stress-induced changes in hypertension and atherosclerosis by antioxidants

Ecarnot (EA3920, University of Franche-Comt and University Hospital Besanon, France) for editorial assistance

doi: 10.1016/j.fsi.2019.07.056 213 NiuK

These data suggest that suppression of NADPH oxidase activity by secreted effector proteins and toxins is not sufficient to defend these bacterial pathogens against oxidative attack and that these factors must work in concert with cellular processes critical for detoxification and repair to prevent restriction by host-derived ROS