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Alterations in brain antioxidant enzymes and redox proteomic identification of oxidized brain proteins induced by the anti-cancer drug adriamycin: implications for oxidative stress-mediated chemobrain

This article provides an in-depth analysis of the molecular mechanisms underlying ferroptosis and delineates its complex interactions with the core pathological hallmarks of AD

Another research in 2018 also reported two special deletions located at positions 3,4183,432 base pair and 10,94110,951 base pair in colorectal cancer [67]

[166] found that the consequence of upregulation of TNF- due to lipopolysaccharide (LPS) stimulation could be blocked by SAMe, MAT, or SAH pretreatment, through increasing the binding of H3K4 to the TNFA promoter