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Studies also suggest that these nutrients support methylation pathways, which are often disrupted in ASD, thereby improving gene expression and reducing oxidative stress

doi: 10.1080/19420862.2015.1007813

demonstrated the in vivo binding of BMAL1 on the human c-Myc promoter and that c-Myc overexpression hindered PER1 transactivation by BMAL1/CLOCK by targeting E-box sequences 31