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2022 Sep 19;14(18):3869

For duplicate CASEIDs, the entry with the latest FDA_DT was retained

A study found that the NRF2-KEAP1 complex keeps shuttling constantly between the cytoplasm and the nucleus, and in the nucleus, the nuclear protein prothymosin competes for NRF2 binding to KEAP1 and enables the liberated NRF2 to activate NRF2 target genes [15]