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Description

Independently assayed HPLC analysis by a third-party lab, per batch

ADAM17-dependent c-METSTAT3 signaling mediates resistance to MEK inhibitors in KRAS mutant colorectal cancer

Its most notable mechanisms include: Promoting angiogenesis (formation of new blood vessels) Reducing pro-inflammatory cytokines Enhancing fibroblast migration and tissue remodeling Supporting gut barrier repair TB-500: The Repair Accelerator TB-500 is a synthetic version of a naturally occurring protein called Thymosin Beta-4 (T4)