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Preclinical evidence shows that 1,25(OH)2D induces a shift from inflammatory (M1) to reparative (M2) macrophages, promoting the secretion of hydrogen peroxide, a critical factor for M.tb clearance [17, 18]

doi: 10.1093/bioinformatics/btv033, 29 LozuponeC.StombaughJ.GordonJ.JanssonJ

In that manner, NMOSD could also be called an astrocytopathy, along with GFAP-IgG autoimmune encephalitis ( Accordingly, AQP4-IgG was demonstrated to induce IL-6 production and release by astrocytes through the NFkB pathway ( While not considered a direct target of disease as in NMOSD, astrocytes retain central roles in other forms of demyelination