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Intriguingly, the diacyl PE-38:4 and PC-36:4 levels are lower in the ferroptosis-sensitive cells (non-NE) than in the ferroptosis-resistant cells (NE) 137

By concurrently activating dual forms of programmed cell death and altering the tumor microenvironment, BB-A-P provides a cohesive and synergistic method for cancer treatment, representing a viable option for addressing hypoxic tumors [9]