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Our own research has shown that GHK promotes the migration of lung fibroblasts from aged mice (24 months old) in a dose-dependent manner (Figure 1), indicating that it can revert aged fibroblasts to a more youthful phenotype, potentially through the activation of mesenchymal progenitor cells

Practical approaches might include targeted delivery to the ovary or testis, combined with standard hormonal protocols, though clinical trials are limited

Hypertrophic and keloid scars fail to progress from the CD34 - /-smooth muscle actin (-SMA) + immature scar phenotype and show gradient differences in -SMA and p16 expression