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In human studies, heteroplasmic germline mtDNA mutations causing complex I dysfunction have shown to shift to homoplasmy in a nasopharyngeal oncocytoma, indicating a selective growth advantage of complex I mutations ( However, no tumor development in the thyroids of Mieap KO/KO and Atg5 thyrKO/KO mice in this study and also in the lungs or colons in the above studies (adenovirus expressing Cretreated Atg5/7 flox/flox and Mieap KO/KO mice) suggests that MIEAP or ATG5/7 KO alone is not sufficient for tumor initiation in the thyroids, lungs, and colons

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Some children may have a harder time recovering from illness or coping with stressful situations