mitochondrial glutathione redox potential Modelling changes in homeostasis as a function of quinone metabolism Thy1-mito-Grx1-roGFP2 mice allow quantification of

diarrhea, constipation, stomach irritation and ulcers, stomach spasm, stomach acid deficiency, gallstones, gall bladder ailments, gout and urinary tract infections

Glutathione undergoes a reaction with the toxic substances, facilitated by enzymes such as glutathione S-transferases, to form a conjugate known as a glutathione adduct, which is then further metabolized in the liver to form mercapturic acid, a water-soluble compound that can be easily excreted through the urine

During the bodys normal functioning, an excess of oxygen-containing molecules is produced

Includes probiotic strains to support gut health after drinking