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However, riboflavin supplementation has not generally been found to increase exercise tolerance or performance (31) unless the individuals are riboflavin deficient (32)

As a well-documented damage-associated molecular pattern (DAMP), HMGB1 engages Toll-like receptors (TLRs) and RAGE, amplifying inflammatory cascades and perturbing endothelial homeostasis[66]

As research progresses, non-histone targets have also been gradually identified, which exhibit a pronounced preference for enzymes involved in metabolic pathways [21]

The supposition that the role of the thymus might involve a hormone-like mechanism led to the isolation from thymus tissue of a biologically active preparation