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In particular, our study showed that the intracellular depletion of GSH is able to induce cellular stress in NO-producing cells through a NO-dependent mechanism, such as inhibition of CcOX activity, DNA damage, and S-NO and 3-nitrotyrosine (NO 2 -Tyr) protein accumulation ( Figure 1 )

Evidence suggests that the autophagy-lysosome system can also contribute to PC elimination to some extent (138140)

Moreover, the ROS activates the NF-kappaB transcriptional factor, which promotes the transcription of various genes, including those involved in the synthesis of nitric oxide and proinflammatory cytokines

Jos A, Trujillo-Hernandez L, Bariat TA, Enders LC, Strader J-P, Reichheld, Christophe Belin (2020) A glutathione-dependent control of the indole butyric acid pathway supports Arabidopsis root system adaptation to phosphate deprivation