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Neurology 50(3):793795 Shults CW, Oakes D, Kieburtz K, Beal MF, Haas R, Plumb S et al (2002) Effects of coenzyme Q10 in early Parkinson disease: evidence of slowing of the functional decline

When tissue folate and cobalamin stores are intact, an abnormal increment of postmethionine-load homocysteinemia may also reveal a saturation of the transsulfuration pathway caused by dietary methionine excess that might be combined with transsulfuration defects (in cases with basal hyperhomocysteinemia) or with the homozygous thermolabile MTHFR defect (in cases with normal fasting homocysteine) (Cattaneo, 1999)

If NO therapy can be introduced in these patients, it may become a promising new treatment strategy