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Results and conclusion To achieve these objectives, the review highlights a significant neuropharmacological knowledge gap, not only regarding less-studied proteins but also concerning key proteins that could ultimately aid in controlling AD, such as Phosphatidylinositol-binding clathrin assembly protein (PICALM), Triggering receptor expressed on myeloid cells 2 (TREM2), Sortilin-related receptor (SORL1), Myc box-dependent-interacting protein 1 (BIN1), and Phospholipid-transporting ATPase ABCA7 (ABCA7), and PD, such as Polyamine-transporting ATPase 13A2 (ATP13A2), Synphilin-1 (SNCAIP), F-box only protein 7 (FBXO7), Septin-4 (SEPTIN4), Neurofilament light polypeptide (NEFL), and Leucine-rich repeat serine/threonine-protein kinase 1 (LRRK1)

(1962) Rat 4h 2850 (1200) 7100 (3000) Audiogenic seizures depressed Frantik et al

First, we analyzed metabolic activity and reactive oxygen species levels to select individual doses for further study

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