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In agreement with that finding and the relative increase of MoAM in chronically smoke-exposed mice, these animals were characterized by impaired lung function as demonstrated by an upward shift of the pressure-volume loop that is typically seen in emphysematous mice ( Figure 6H ) ( Figures 6I, J )

After long-term exposure to cigarette smoke, germinal centers with T cells and B-cell zones may form at the site, supporting the production of pathogenic autoantibodies and driving autologous disease