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Given that implanted chondrocytes are responsible for the generation of new cartilage in the defect site, it is not surprising that the quality of these cells critically affects the reparative results of ACI treatment (Davies and Kuiper, 2019)

This condition consists of depleted iron stores with ferritin levels lower than 10 mcg/L, plus an RBC count falling below normal range with hemoglobin below 12 g/dL and hematocrit below 36% for women and 41% for men

Although initial studies suggested that GSH depletion was only a byproduct of oxidative stress generated during cell death, recent discoveries suggest that GSH depletion and post-translational modifications of proteins through glutathionylation are critical regulators of apoptosis

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