We hypothesized that high 15LO1, PEBP1, and GPX4 activity drives abnormal asthmatic redox biology, evidenced by lower bronchoalveolar lavage (BAL) fluid and intraepithelial cell GSH:oxidized GSH (GSSG) ratios, to enhance type 2 (T2) inflammatory responses
Interestingly, Gsta4, the main scavenger of the lipid peroxidation product 4-hydroxynonenal (4-HNE), has been predicted to be a target of Nrf2 51,52 , which we corroborated through the observation that both DMF and Clem-F induced the expression of Gsta4 (Fig
Similar content being viewed by others Introduction Endometriosis is a chronic, estrogen-dependent, and inflammatory gynecological disorder characterized by the ectopic growth of endometrial-like tissue outside the uterine cavity [1, 2]
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