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Simultaneously, ALP, which generally maintains cellular homeostasis by degrading damaged organelles, is pathologically increased in CC, contributing to muscle atrophy through the degradation of both fiber proteins and organelles (Sandri, 2013)

For example, retinoid X receptor ( RXRG ) is targeted by gga-miR-140-5p, whereas sterol carrier protein 2 ( SCP2 ), acetyl-CoA acyltransferase 1 ( ACAA1 ), and acyl-CoA oxidase 1 ( ACOX1 ) are targeted by miR-15a to promote the differentiation of intramuscular preadipocytes, thereby increasing IMF deposition in muscle [78, 79]