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Emerging clinical evidence suggests that the SARS-CoV-2 virus can directly hijack or damage mitochondrial networks during acute infection, leaving a legacy of profound metabolic dysfunction long after the initial virus has been cleared

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Conclusions ROSs play an essential role in the autophagy-associated apoptosis in PFOS-treated RTCs, which is associated with the impairment of mitochondrial bioenergetics and the effects of uncoupling on reduced ATP synthesis