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Surprisingly, there was not a noticeable cell loss in the culture of non-senescent PDL3 chondrocytes, suggesting the selective potential of FOXO4-DRI

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This paradox may be explained by the increased insulin resistance and the compensatory intra-portal hyperinsulinemia causing increased hepatic GH sensitivity that increases serum IGF-I and blunts pituitary GH secretion via the negative feedback loop