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36 It is possible that the inhibition of liver regeneration after Tnfr1 deletion in the latter study 36 could have been mediated secondary to exaggerated injury, which needs further exploration

To generate non-site-specifically acetylated TrxR1, 500 nM TrxR1 was incubated with increasing concentrations (015 mM) of aspirin (A5376-250G, Sigma) in phosphate buffer for 1 h at 37C

This discrepancy likely stems from the complex nature of microbial MK, where microbiological evaluation remains crucial for accurately identifying the causative organism, as the clinical features of corneal ulcers are not always diagnostic of the underlying pathogen

doi: 10.1186/s12896-017-0409-7 67 Garcia-ManteigaJMClarelliFBonfiglioSMasciaEGianneseFBarbieraGet al