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Theyre tools, and like any tool, their value depends entirely on how intentionally you use them

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Taking all this data together it seems logical or appropriate to conclude that, because IGF1-R has a different signalling pathway that can maintain lipid oxidation in the liver, FFA uptake in muscle, and activates mTORC1 could reactivate IR through tyrosine kinase activity on IRS, thus displacing serine phosphorylation, reinstituting insulin signalling

Why Standard Tests Can Be Misleading This brings up a critical point that resonates with so many people who feel their symptoms are being dismissed