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One of the MGO adducts, N --(carboxyethyl)-lysine (K ce hereafter, shown in Fig

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Researchers have identified that heterozygous functional loss mutations in the KCNK3 gene (member 3 of the potassium channel subfamily K) are responsible for hereditary PAH, as KCNK3 encodes an outwardly rectifying potassium (K +) channel [87, 88]