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In: Thorac Surg Clin

doi: 10.3390/ijms241813784

Together, these pathways are of interest in models evaluating energy expenditure , mitochondrial stress adaptation, and body-composition endpoints

Specifically, CLP-induced systemic inflammation may activate microglia to secrete pro-inflammatory mediators, which suppress the Nrf2/HO-1/GPX4 redox axis, triggering mitochondrial dysfunction and oxidative stress