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The hydroxylated HIF-1 is recognized by the von Hippel-Lindau (VHL) E3 ubiquitin ligase complex, leading to its ubiquitination and subsequent rapid degradation via the proteasome pathway, thus maintaining extremely low levels of HIF-1 under normal oxygen tension (Maxwell et al., 1999

Of note, TCE-mediated skewing of hepatic and splenic immune lineage development was effectively reversed by NAC (Table 1)

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Blood coagulation in Prediabetes clusters-impact on all-cause mortality in individuals undergoing coronary angiography