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A recent study in neonatal murine models found that hyperoxia reduces cilia length, decreases motor proteins, and causes mitochondrial dysfunction in ciliated cells [65]

The A /Cu complex required ~0.75 mole metal per mole of peptide for successful inhibition of the enzymes activities, thus suggesting the formation of a reactive by-product (from the complexation) that reacted to cytochrome c oxidase [221-223]

During their migration into the circulatory system, these cells may also face ROS-enhanced oxidative stress (Nieto et al