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Pathologically, alveolar epithelial and pulmonary microvascular endothelial cells are damaged, and the permeability of microvascular basement membrane increases, leading to fluid accumulation in the interstitium or alveolar cavity [103]

These data suggest gemcitabine alone induced EMT, while inhibition of both HGF and c-MET (Hi + Ci) protected from EMT

Inhibiting recruitment and expansion of MDSCs The recruitment and accumulation of MDSCs in the TME create an immunosuppressive microenvironment, leading to poor clinical outcomes and therapy resistance