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Ionizing radiation promotes the acquisition of a senescence-associated secretory phenotype and impairs angiogenic capacity in cerebromicrovascular endothelial cells: role of increased dna damage and decreased dna repair capacity in microvascular radiosensitivity

MtDNA damage results in impaired ETC function, reduced ATP generation, increased levels of ROS and disrupted calcium homeostasis, leading to exacerbated amyloid- (A) processing and aggregation in AD mice (47)

The primary cause of INH-induced hepatotoxicity is in vivo metabolism involving biotransformation on its terminal -NH 2 group owing to its high nucleophilic nature