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Despite their historic popularity and effectiveness with nearly every disease, they have not been studied nearly as much as you would expect

Mechanistic insights from non-AD neuroinflammatory systems provide complementary evidence: interferon- (IFN-) robustly induces PD-L1 expression in human astrocytes and microglia, establishing a checkpointredox interface that suppresses excessive oxidative injury (Smith et al., 2023

Another consideration is whether to target intracellular or extracellular (secreted) expression

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