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Within this integrated model, UA-driven mitophagy initiation, ALA-supported flux completion, and EGT-mediated mitochondrial stabilization converge to modulate the ferroptosismitophagy regulatory axis in cardiometabolic pathology through complementary mechanisms

Increased characterization and identification of novel PTMs that regulate the pathophysiological functions of RBPs will improve our ability to discern the pathological and physiological characteristics of RBPs in the development and progression of ALS