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Studies in the MRL/lpr lupus-prone mouse model have demonstrated that gut dysbiosis can induce intestinal oxidative stress, compromise the gut barrier (evidenced by reduced ZO-2 and elevated fecal albumin and IgA), promote bacterial translocation, and exacerbate systemic inflammation and autoimmune responses [37]

Also, it has to be kept in mind that these assays, structures are of NET in a detergent micelle, so an important portion of membrane physiology is missing in these (membrane