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Taken together, these studies underscore the therapeutic potential of iPSC-derived neural progenitors in PD models, yet the variability in reprogramming methods reported in the aforementioned studies, limited scalability, and unresolved concerns around long-term safety and functional integration highlight the need for more standardized and clinically validated approaches

Genome-wide identification of the WRKY transcription factors family and regulation of metabolites under cold stress in Astragalus membranaceus

Pharmacologic MCU inhibition restores autophagy and immune function in CF models, highlighting MCU as a promising target to modulate inflammation [194]