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[ 404 ] The deletion of BACH1 reversed the Pbmediated transcriptional and expressive decrease of genes associated with oxidative phosphorylation (OXPHOS) and eliminated the growth suppression of axon activation and synapse formation induced by Pb

a , Immunofluorescence staining of HeLa cells transfected with in vitro transcribed mRNA encoding DARPin C10 (upper row) or control DARPin (cDP) (lower row)

Fig 4B)

These researchers further demonstrated that this synaptic dysfunction was caused by these human A42 dimeric oligomers by completely blocking the disruption of synaptic plasticity by passive immunization with antibodies to A42 dimers