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The vascular endothelium and cavernosal arteries are a source of vasorelaxing factors such as NO, prostaglandin I2, endothelium-derived hyperpolarizing factor (EDHF), the vasoconstrictor factors such as angiotensin II (Ang II) and endothelin-1 (ET-1), cell adhesion molecules such as Intercellular Cell Adhesion Molecule-1 (ICAM-1) and Vascular Cell Adhesion Molecule-1 (VCAM-1), growth factors such as vascular endothelial growth factor (VEGF), platelet-derived growth factor (PDGF), inflammatory factors, etc

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Mechanisms of NO dysregulation NO deficiency can occur through mechanisms such as eNOS uncoupling, abnormal phosphorylation, inhibition by endogenous methylarginines, and increased NO degradation due to ROS

MehrzadJZahraei SalehiTKhosraviAHosseinkhaniSTahamtaniYHajizadeh-SaffarEet al