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Examined in oxidative stress response models: Oxidative stress contributes to telomere shortening and cellular damage

By the time most patients are in their late 30s or 40s, pineal calcification and reduced melatonin synthesis have already begun degrading sleep architecture, particularly the slow-wave deep sleep phases where cellular repair, growth hormone release, and memory consolidation occur

Research in aging rats demonstrated that Epithalon treatment significantly increased activities of key antioxidant enzymes including superoxide dismutase (SOD), glutathione peroxidase (GPX), and glutathione-S-transferase