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Description

Chalvin et al., 2020)

Future research should concentrate on adapting these synergistic regimens to the specific pathophysiology of individual AIDs, thereby creating the basis for personalised cellular therapies

Aspergillus flavus, Aspergillus fumigatus, Aspergillus niger, Aspergillus terreus, Bacillus cereus, Bacillus megaterium, Bacillus subtilis, Candida albicans, Candida glabrata, Candida krusei, Candida parapsilosis, Candida sake, Candida tropicalis, Enterococcus faecalis, Escherichia coli, Klebsiella oxytoca, Klebsiella pneumoniae, Listeria monocytogenes, Micrococcus luteus, Paecilomyces variotii, Penicillium expansum, Pityriasis alba, Proteus mirabilis, Proteus vulgaris, Pseudomonas aeruginosa, Pseudomonas fluorescens, Rhizopus oryzae, Salmonella enterica, Salmonella typhi, Serratia marcescens, Shigella dysenteriae, Shigella flexneri, Shigella sonnei, Staphylococcus aureus, Serratia marcescens, Staphylococcus epidermidis, Staphylococcus hominis, Streptococcus mutan, Pasteurella multocid, Vibrio parahemolyticus , and Vibrio cholerae