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Oxidative stress has been increased as a result of mitochondrial dysfunction, endothelial nitric oxide synthase (eNOS) uncoupling, resulting in endothelial dysfunction, the aberrant activity of NADPH oxidases (NOX), suppressed activity of antioxidant enzymes and low molecular weight antioxidants (Galvan et al

Indeed, we find that the distribution of 13-yr DJFM TX10d trends in observations is wider and significantly different from that of the 500-yr FLOR simulation (Supplementary Note 5 and Supplementary Fig

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