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Description

Exosomes Derived from Baicalin-Pretreated Mesenchymal Stem Cells Alleviate Hepatocyte Ferroptosis after Acute Liver Injury via the Keap1-NRF2 Pathway

Table 1 Therapeutic mechanisms and evidence-based effects of key nutrients associated with OA

Fino2 initiates ferroptosis through gpx4 inactivation and iron oxidation

First, the causal direction in human studies remains ambiguous: it is unclear whether gut dysbiosis is a driver of OA or a consequence of its associated factors, such as reduced physical activity, diet changes, or chronic medication use