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By contrast, non-canonical NF-B signaling plays an important role in the expression of proteins that facilitate Tfh differentiation, such as ICOS ligand (ICOSL) in BAFF-stimulated B cells (99)

These phenotypes are similar to those detected in mice harboring NIK deficiency or non-functional mutations [3, 4, 6, 67], suggesting that the immunoregulatory function of NIK is remarkably conserved between humans and mice

The HES-like inflammatory disease in NIK-deficient mice involves increased production of cytokines by Th2 subset of CD4 + T cells via a nonautonomous mechanism that depends on radiation-resistant non-hematopoietic cells

These data indicate that NTAPP promotes hair growth in mice via activation of the -catenin signaling pathway in murine skin (graphical summary in Fig