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Alpha-synuclein oligomers interact with metal ions to induce oxidative stress and neuronal death in Parkinsons Disease

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Thus, T4 has substantial potential for clinical translation as a multiple-target therapy for MS/EAE or other neurological demyelinating diseases, with the remyelination effect supplementing its anti-inflammatory and neuroprotective role, as previously found in studies involving T4 prophylactic treatment (Zhang et al., 2009)