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Silencing GPX3 essentially nullifies the antioxidant effects of TZDs, indicating that the PPAR-dependent antioxidant effects of these drugs are mediated through GPX3 [39]

System x c was reported to modulate both systemic and central inflammation [4, 5], and xCT expression can be induced by typical hallmarks of aging, i.e., inflammation and oxidative stress [9]

In this setting, HPV-16 E7 Mut expression was only able to partially protect HaCaT cells from death (Figure 7B, bars 1 and 5), and this effect was drastically reduced in GSTP1-silenced cells (Figure 7C, bars 5 and 6)

For a more detailed description of this algorithm and a substantiation of the statistical approach used, see Supplementary Note 1