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Excess glutamate leads to over-stimulation of postsynaptic glutamatergic receptors, particularly NMDARs and AMPARs, allowing calcium to enter the cell, causing neuronal depolarisation and further neuronal death (Glotfelty et al., 2019), Inhibition of glutamate release, enhancement of glutamate clearance and blockade of glutamate receptors may be major directions for future stroke research