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This review summarizes the pathways involved in ferroptosis resistance in GC cells and the mechanisms underlying the therapeutic effects of herbal formulae and their bioactive compounds, with particular emphasis on ferroptosis

Acknowledgements We thank Michael Sporn for supplying CDDO TFEA , Naomi Fukagawa and Shyam Biswal for plasmids and Andreas Strasser for the Puma-null founder mice supplied to the laboratory of Aviva Tolkovsky and utilized by her coworker Michael Fricker (co-author)

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BPC-157 research has focused on its interactions with the nitric oxide (NO) system, growth factor modulation (including VEGF, FGF-2, and EGF receptor pathways), and FAK-paxillin signaling in cell migration studies