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Thus, in this review we will discuss the involvement of Nrf2 in the mechanisms of action of classic analgesic and anti-inflammatory drugs, as well as natural products and other molecules that modulate Nrf2, in the context of experimental models (summarized in Table 1) and human diseases (summarized in Table 2)

There are specific formulations for oral use that allow for better stability and prevent degradation

Trizio, C

DNA hypermethylation inactivates tumor-suppressor genes such as adenomatous polyposis coli (APC), retinoblastoma (Rb), or BRCA1, as well as others involved in DNA repair such as MGMT, apoptosis (DAPK) or antioxidation (GSTP1) [8, 9]