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Gerussi A, Natalini A, Antonangeli F, Mancuso C, Agostinetto E, Barisani D, et al

Birko S, Ravitsky V, Dupras C, et al

Evidence suggests: Normal telomerase activation cancer risk Key difference: Cancer has multiple mutations beyond telomerase Normalized telomerase in healthy cells appears safe Animal study cancer data: Mechanisms of cancer protection: Immune system enhancement detects and eliminates pre-cancerous cells Antioxidant effects reduce DNA damage (cancer initiation) Normal cell cycle regulation vs dysregulation Apoptosis (programmed cell death) in damaged cells Reduced chronic inflammation (cancer promoter) Important caveats: Animal data doesn't guarantee human cancer prevention No human cancer prevention trials completed Not a substitute for medical cancer screening Should not delay conventional cancer treatment Theoretical benefit needs validation Learn about peptide safety and research at SeekPeptides

5.2 Suppression of pro-inflammatory signaling (NF-B and MAPKs) Several organosulfur compounds (OSCs), including allicin, ajoene, and diallyl disulfide (DADS), attenuate canonical pro-inflammatory signaling by targeting redox-sensitive components of upstream kinase pathways