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KhurelchuluunAUeharaOPaudelDMorikawaTKawanoYSakataMet al

Subsequently, multiple pro-fibrotic factors (e.g., chemokines, proteases, and TGF-) are released, leading to the transition of AEC-II into a mesenchymal phenotype through the process of epithelial-mesenchymal transition (EMT), ultimately resulting in basement membrane destruction [8, 37]

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Results and discussion Considering MS cases have been increasing worldwide and few drug choices are available, this study aimed to identify potential activators of GPx4, an essential inhibitor to ferroptosis, as a novel neuroprotective strategy in MS treatment through computational technologies