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The most typical manifestation is impaired aerobic endurance exercise performance, with adolescent and female athletes being particularly prone to iron deficiency

Several reports revealed that synovial macrophage pyroptosis promotes OA inflammation in chondrocytes through cytokine secretion (e.g., IL-1, IL-18), EV release with bioactive molecules, and direct chondrocyte death induction, leading to cartilage damage and OA progression[39], [40], [41]

In its active form (quinol), coenzyme Q10 can scavenge several ROS or regenerate other oxidized antioxidants (including vitamins C and E)

There are other Phase 2 enzymes and proteins that perform similar functions, but without glutathione, these other enzymes couldnt adequately function